What Does Hcp Stand For In Healthcare?

What Does Hcp Stand For In Healthcare
Acronyms and Abbreviations

Abbreviation	Expansion
HCP	Healthcare Personnel
HICPAC	Healthcare Infection Control Practices Advisory Committee
HIPPA	Health Insurance Portability and Accountability Act
HIV	Human Immunodeficiency Virus

19 more rows

What does HCP mean in therapy?

A person with a High Conflict Personality (HCP) usually has underlying trauma associated with distinct personality traits.

What is the role of the HCP?

Healthcare Professionals (HCPs) provide medical care and, when required, forensic assessment of detainees/suspects in police custody, complainants/complainers of crime, police officers/ police personnel injured in the course of their work.

What is HPC in medical terms?

Hematopoietic progenitor cells (HPC) from mobilized peripheral blood display enhanced migration and marrow homing compared to steady-state bone marrow HPC – PubMed Display options Format Abstract PubMed PMID Objective: Faster engraftment of G-CSF-mobilized peripheral blood (MPB) transplants compared to steady-state bone marrow (ssBM) is well documented and clinically relevant. A number of different factors likely contribute to this outcome. In the present study we explored whether independent of cell number there are intrinsic differences in the efficiency of progenitor cell homing to marrow between MPB and ssBM. Methods: Mobilization was achieved by continuous infusion of G-CSF alone or in combination with other mobilizing agents. In vivo homing assays, in vitro migration assays, gene expression analysis, and flow cytometry were utilized to compare homing-related in vivo and in vitro properties of MPB and ssBM HPC. Results: Marrow homing of murine MPB HPC, generated by different mobilizing schemes, was reproducibly significantly superior to that of ssBM, in lethally irradiated as well as in nonirradiated hosts. This phenotype was independent of MMP9, selectins, and beta2- and alpha4-integrins. Superior homing was also observed for human MPB HPC transplanted into NOD/SCIDbeta2microglobulin(-/-) recipients. Inhibition of HPC migration abrogated the homing advantage of MPB but did not affect homing of ssBM HPC, whereas enhancement of motility by CD26 inhibition improved marrow homing only of ssBM HPC. Enhanced SDF-1-dependent chemotaxis and low CD26 expression on MPB HPC were identified as potential contributing factors. Significant contributions of the putative alternative SDF-1 receptor, RDC1, were unlikely based on gene expression data. Conclusion: The data suggest increased motility as a converging endpoint of complex changes seen in MPB HPC which is likely responsible for their favorable homing. Figure 1 Superior marrow homing of MPB Figure 1 Superior marrow homing of MPB grafts: A-D: In vivo marrow homing of murine Figure 1 Superior marrow homing of MPB grafts: A-D: In vivo marrow homing of murine B6/129 CFU-C from ssBM or MPB was quantified, using colony assays. A: Three hours after transplantation of lethally irradiated hosts, the mean fraction of CFU-C homed to bone marrow was 41% greater for MPB than for ssBM (*p<0.05). B: A similar marrow homing advantage as for G-CSF-MPB was observed after 18 hours for CFU-C mobilized with Cyclophosphamide+G-CSF(*p<0.001) or with Flt3L+G-CSF (not shown). C: Forty-eight hours after transplantation, recovery from recipient marrow of MPB-derived CFU-C was significantly greater compared to ssBM-derived CFU-C. This observation was made in both irradiated (left, *p<0.005) and nonirradiated (right, *p<0.005) hosts, ruling out a role of the irradiated marrow microenvironment in the differential marrow homing of ssBM and MPB. For both MPB and ssBM, the fraction of homed CFU-C was markedly increased 48 hours after transplantation compared to 18 hours, likely indicating early proliferation of the transplanted cells. Depicted in Fig.1A-C are mean+SEM for % of injected CFU-C homed to marrow (MPB, ssBM ). D: Forty-five% more efficient mean marrow homing of MPB CFU-C compared to ssBM CFU-C was observed 18 hours after transplantation of lethally irradiated isogeneic recipients (p<0.001). Each dot represents homing in one individual animal; bold line and error bars represent mean±SEM for % of injected CFU-C homed to marrow. E: Marrow homing of human CD34+ cells from ssBM or MPB was tested in a xenotransplantation model. Eighteen hours after transplantation, the mean recovery of MPB CFU-C from recipient marrow was 3-fold increased compared to ssBM (*p<0.001). Each dot represents homing in one individual animal; bold line and error bars represent mean±SEM for % of injected CFU-C homed to marrow. These data indicate that the greater marrow homing ability of MPB CFU-C is intrinsic to a population enriched for HPC, and that it applies to human as well as to mouse CFU-C. Figure 2 Increased migration of MPB cells: Figure 2 Increased migration of MPB cells: Transwell migration of MPB c-kit+ cells (A), Figure 2 Increased migration of MPB cells: Transwell migration of MPB c-kit+ cells (A), both spontaneous and SDF-1 directed, was markedly greater than that of ssBM c-kit+ cells (*p<0.005 MPB vs. ssBM), and SDF-1 directed migration of MPB-CFU-C (B) was likewise increased (*p<0.005). CFU-C did not show appreciable spontaneous migratory activity (not shown). Superior chemotactic migration was observed despite decreased CXCR4 expression (p<0.005) on MPB c-kit+ cells. Shown is one representative histogram plot ( C ). Incubation with Pertussis toxin (PTX) blocked spontaneous and chemotactic migration of MPB (left) and ssBM (right) c-kit+ cells ( D ). Gene expression of the alternative SDF-1 receptor, RDC1, in MPB MNC was very low, since it was only detected after 40 PCR cycles, which makes a contributory role of RDC1 to MPB HPC migration unlikely ( E ). Migration is depicted as mean+SEM of % of total. Cell type is indicated in the upper right corner, cell source and stimulus below the column. The FACS histogram depicts expression of CXCR4 on c-kit+ cells (Isotype control lightly shaded, MPB black line, ssBM solid grey; cell counts on Y-axis, fluorescence intensity on X-axis). Figure 3 Increased migration is responsible for Figure 3 Increased migration is responsible for superior marrow homing of MPB CFU-C: A: Cartoon Figure 3 Increased migration is responsible for superior marrow homing of MPB CFU-C: A: Cartoon depicting the experimental approach: The consequences of altered CFU-C migration for marrow homing were tested. This was done by blocking migration through inhibition of CXCR4 or Gi protein signaling (using AMD3100 or PTX), and by increasing migration (using DiprotinA, an inhibitor of the dipeptidylpeptidase CD26). B-D: AMD3100 (marrow homing assessed 3 hours after transplantation, B ) and PTX (marrow homing assessed 18 hours after transplantation, C ) significantly blocked marrow homing of MPB CFU-C (*p<0.005 compared to untreated MPB), but had no effect on ssBM homing. In contrast, DiprotinA significantly increased marrow homing only of ssBM CFU-C (*p<0.05 compared to untreated ssBM, marrow homing assessed 18 hours after transplantation, D ). No DiprotinA effect on MPB homing was observed, likely due to loss of CD26 expression on MPB c-kit+ cells ( E ). These data suggest that the more avid migration of MPB cells, mediated through SDF-1, CXCR4 and Giprotein signals, guides their better marrow homing. Homing data are shown as mean+SEM for % of injected CFU-C homed to marrow, (MPB, ssBM, solid: - inhibitor, hatched: + inhibitor; * indicates significant difference (p<0.05) compared to samples from the same source that were not treated with the inhibitor). The FACS histogram depicts expression of CD26 on c-kit+ cells (Isotype control lightly shaded, MPB black line, ssBM solid grey; cell counts on Y-axis, fluorescence intensity on X-axis). Figure 4 Favorable homing of MPB persists Figure 4 Favorable homing of MPB persists in the absence of MMP9, β2-integrin and selectins: Figure 4 Favorable homing of MPB persists in the absence of MMP9, β2-integrin and selectins: Previously reported differential regulation of MMP-9, β2-integrins and selectins between MPB and ssBM HPC prompted us to test their contribution to marrow homing, using genetically deficient donors or recipients. Marrow homing of MPB or ssBM CFU-C from MMP-9-/-mice ( A ) CD18-/-mice ( B ) or CD62L-/-mice ( D ) in WT recipients or of WT CFU-C in CD62ELP-/-recipients ( C ) was entirely normal, i.e. no different than marrow homing in WT-to-WT transplantation, as shown in Fig.1. The superior marrow homing of MPB-derived CFU-C was maintained (*p<0.005, p<0.05, p<0.001, p<0.005, respectively for ssBM vs. MPB). Eighteen hour homing data are shown as mean+SEM for % of injected CFU-C homed to marrow (MPB, ssBM ).

What does HCP stand for Neuro?

Abstract – Background: Hereditary Coproporphyria (HCP) is characterized by abdominal pain, neurologic symptoms and psychiatric disorders, even if it might remain asymptomatic. The pathophysiology of both neurologic and psychiatric symptoms is not fully understood.

Therefore, aiming to evaluate a possible role of brain blood flow disorders, we have retrospectively investigated cerebral perfusion patterns in Single Photon Emission Computed Tomography (SPECT) studies in HCP patients.
Materials & Methods: We retrospectively evaluated the medical records of patients diagnosed as being affected by HCP.

A total of seven HCP patients had been submitted to brain perfusion SPECT study with 99mTc-Exametazime (hexamethylpropyleneamine oxime, HMPAO) or with its functionally equivalent 99mTc-Bicisate (ECD or Neurolite) according with common procedures. In 3 patients the scintigraphic study had been repeated for a second time after the first evaluation at 3, 10 and 20 months, respectively.

All the studied subjects had been also submitted to an electromyographic and a Magnetic Resonance Imaging (MRI) study of the brain.
Results: Mild to moderate perfusion defects were detected in temporal lobes (all 7 patients), frontal lobes (6 patients) and parietal lobes (4 patients).
Occipital lobe, basal ganglia and cerebellar involvement were never observed.

In the three subjects in which SPECT study was repeated, some recovery of hypo-perfused areas and appearance of new perfusion defects in other brain regions have been found. In all patients electromyography resulted normal and MRI detected few unspecific gliotic lesions only in one patient.

Discussion & Conclusions: Since perfusion abnormalities were usually mild to moderate, this can probably explain the normal pattern observed at MRI studies. Compared to MRI, SPECT with 99mTc showed higher sensitivity in HCP patients. Changes observed in HCP patients who had more than one study suggest that transient perfusion defects might be due to a brain artery spasm possibly leading to psychiatric and neurologic symptomatology, as already observed in patients affected by acute intermittent porphyria.

This observation, if confirmed by other well designed studies aiming to demonstrate a direct link between artery spasm, perfusion defects and related symptoms could lead to improvements in HCP treatments. Keywords: Artery spasm, Hereditary Coproporphyria, perfusion defect, porphyrias, porphyric attack, SPECT (Single Photon Emission Computed Tomography)

What is HCP assessment?

Background –

CoP = Conditions of Participation
HCO = Healthcare Organization
HCP = Healthcare Personnel
IPC = Infection Prevention and Control
NHSN = National Healthcare Safety Network
OHS = Occupational Health Services
OSHA = Occupational Safety and Health Administration
PPE = Personal Protective Equipment
TB = Tuberculosis

HCP are at risk of infectious exposures in the workplace that vary depending on their job duties and other factors. Assessments can be conducted to identify actual or potential infection risks for populations of HCP and to inform measures that reduce those risks.

What does HCP engagement mean?

Digital Evolution – Effective digital participation in most industries necessitates a considerable amount of change management. A well-coordinated digital plan means that the right new processes are designed, reviewed, and streamlined to help the company grow.

Each company’s speed and magnitude of transition can vary based on the current landscape, history, and internal capabilities. For commercial success, the pharma industry needs to harness digital evolution and adopt new and innovative ways to access their customer base, which will deliver a broader channel mix.

In addition, pharma sales reps should be multi-channel communicators who put their customers’ preferences at the heart of every sales interaction. Build your HCP engagement through the expert services of today. You can contact us by emailing us at or calling, What Does Hcp Stand For In Healthcare Looking for more information? Have a browse through our Frequently Asked Questions to see what our other clients are enquiring about. If you have further questions, please don’t hesitate to get in touch with one of our experts using the Talk to Us form at the bottom of this page.

Pharma HCP engagement is the process of delivering relevant, pertinent, and timely medical information to healthcare providers by the industry that serves them. This includes information from pharmaceutical companies regarding new drugs on the market, their applications, and their side effects. Traditionally, engagement was largely carried out face-to-face by sales reps, but increasingly, engagement for healthcare professionals online has become more effective – and cost-effective too.

HCP stands for healthcare professionals (HCPs). This term includes doctors (both generalists and specialists), nurses, midwives, pharmacists, dentists, and any others who work in the healthcare profession and seek to maintain health through the application of evidence-based medicine and caring.

In terms of marketing, communicating with these individuals is increasingly done through a healthcare professional engagement platform. The definition of HCP (Healthcare Professional) includes any individual who studies, diagnoses, treats or prevents illness or injury in humans; advises on or applies preventative or curative treatments; researches methods to improve evidence-based healthcare; and/ or promotes health in individuals or populations to improve health outcomes.

In the past, healthcare professional collaboration was primarily face-to-face through meetings between HCPs and pharmaceutical sales representatives. Nowadays, engagement through digital channels is more appropriate. These methods may include virtual HCP engagement sessions; email campaigns; digital newsletters; direct mail marketing; and cloud-based software platforms.

These methods are seen to be more customer-centric and can reach wider audiences in the most cost-effective manner. To help HCPs perform their roles effectively, HCP engagement for the pharma industry must involve up-to-date information, education, training, and discussion on the latest research. The pharmaceutical industry works with HCPs to develop and deliver medicines that can improve patient health outcomes.

Healthcare professional engagement also supports improvements in developing medicines, as HCPs can offer first-hand experience of patient care. Segmentation refers to the process of dividing up your target audiences into different segments by using certain agreed criteria and targeting the most relevant information in the most relevant way possible to each segment.

This improves healthcare professional interaction as it increases users’ engagement with particular marketing campaigns or messages because your information is tailored more accurately to their needs. You need HCP engagement platforms and programmes that make it easier for HCPs to navigate an exceedingly complex healthcare system in order to target the right people with the best services available.

Consider these factors when coming up with HCP engagement solutions:

Audiences should be segmentedPersonalised experience reigns supremeMake as much use of digital pharma HCP marketing as possibleMailers can compensate for sales reps’ limited access to physicians

: HCP Engagement – Effective Marketing | Star OUTiCO

What is HCP in business?

HCP marketing in today’s digital landscape – Healthcare professional marketing—or HCP marketing—is all about creating smart customer segmentation and targeted campaigns that healthcare companies and the pharmaceutical industry can leverage to drive usage across a product’s lifecycle.

The COVID-19 pandemic had a significant impact on HCP marketing, meaning physician engagement is now more complex than before and demands an increasingly digital approach.
Social media engagement, in particular, has skyrocketed,
We’ve seen this ring true on our own physician social platform: throughout the COVID-19 pandemic, physicians sharing their drug perceptions through Sermo’s Drug Rating platform increased by nearly 70%, and participation in online social polls skyrocketed by almost 300%.

Who are HCP in pharma?

What Does HCP Stands for in Pharma Marketing? – The HCP meaning in pharma has different interpretations. HCPs stand for healthcare professionals, providers, or practitioners. HCPs can be individuals like physicians, dentists, nurses, pharmacists, or institutions like hospitals, clinics, and nursing homes.

What does HPC stand for in nursing?

Health Providers Choice. Welcome! This is the place where you can find travel nurse practitioner jobs and learn about building your travel nurse resume.

What does HPC mean job?

Supercomputing is an appealing career path for anyone in IT who wants to work on exciting, demanding projects that make a difference in the world. Here are the skills you need to find a job in this growing field. IT professionals who are interested in working on the leading edge of computing should learn about supercomputers and high-performance computing (HPC).

This field of activity encompasses a variety of job roles that range from the people who specify and build the systems, to the admins and engineers who put them together and run them, to the programmers who create the tools and applications that put the monsters to work. One thing they all have in common, though: HPC work is demanding, exciting, and often has an immediate effect on the world.

Predicting the weather? Sequencing genomes to help researchers solve Alzheimer’s ? Designing industry-leading special effects for a movie ? Few of these uses for supercomputers are boring. Here’s a short technical overview of HPC, followed by career options in this super-fast segment of the computer market.

Who are HCP in pharma?

What does HCP stand for Neuro?

Therefore, aiming to evaluate a possible role of brain blood flow disorders, we have retrospectively investigated cerebral perfusion patterns in Single Photon Emission Computed Tomography (SPECT) studies in HCP patients.
Materials & Methods: We retrospectively evaluated the medical records of patients diagnosed as being affected by HCP.

All the studied subjects had been also submitted to an electromyographic and a Magnetic Resonance Imaging (MRI) study of the brain. Results: Mild to moderate perfusion defects were detected in temporal lobes (all 7 patients), frontal lobes (6 patients) and parietal lobes (4 patients). Occipital lobe, basal ganglia and cerebellar involvement were never observed.

Discussion & Conclusions: Since perfusion abnormalities were usually mild to moderate, this can probably explain the normal pattern observed at MRI studies. Compared to MRI, SPECT with 99mTc showed higher sensitivity in HCP patients. Changes observed in HCP patients who had more than one study suggest that transient perfusion defects might be due to a brain artery spasm possibly leading to psychiatric and neurologic symptomatology, as already observed in patients affected by acute intermittent porphyria.